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methylphenidate) in ADHD, we hypothesize that the disruption of D2R-DAT protein complex might be a new treatment means for ADHD by enhancing the dopaminergic neurotransmission

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256 Mutations in Fbw7, observed in breast and ovarian cancers, lead to cyclin E accumulation, premature S-phase entry, chromosomal instability, and tumor formation

in addition, GSH becomes not just an inhibitor of inflammation but a regulator of innate immunity in a direction that benefits the host (134)

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